0:00:10 | i met a problem and i've been working in campus lab to the u burke |
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0:00:14 | program and we are studying h p v or human couple on the virus type |
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0:00:18 | sixteen |
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0:00:19 | and h p v types sixteen is the main cause of cervical cancer it causes |
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0:00:24 | fifty percent of all cervical cancers each year |
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0:00:30 | all the other h p v types combined account the other fifty percent |
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0:00:34 | and the question we wanted to solve when it comes h p v infection |
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0:00:37 | is how does invade a whole cell and set up a success one faction |
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0:00:43 | and h p v is a non on the left virus and so when it |
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0:00:47 | gets taken into a cell |
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0:00:49 | it encounters a serious there you're to its successful infection in the form of an |
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0:00:55 | in this normal member |
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0:00:56 | when it gets taken in |
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0:00:58 | it has to get its genome across the non polar |
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0:01:03 | and is a membrane this is a difficult process because the aging on is very |
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0:01:06 | pull |
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0:01:08 | in order to do this the |
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0:01:10 | the virus somehow has to disrupt that membrane or for a pork process |
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0:01:15 | we don't know which way h p v is doing whether just completely destroying the |
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0:01:20 | and dissolve or if it's forming a small or through which can get it you |
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0:01:24 | know so we found this trend membrane region on that caps approaching |
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0:01:29 | when we looked closer at it we found these highly conserved g x three g |
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0:01:32 | motives all throughout the trans membrane domain |
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0:01:36 | g extra g motifs |
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0:01:38 | are |
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0:01:39 | protein multi switch in which there's one nikolai seen then three of any amino acid |
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0:01:44 | then another glancing |
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0:01:45 | these motives of previously been shown to mediate trans membrane domain interactions within membrane so |
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0:01:52 | they the question is an extremely small amino acid |
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0:01:56 | so what that what these three g motif do is on alpha helix |
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0:02:02 | puts the glide things right on top of each other and sense that pricing is |
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0:02:05 | so small it provides almost like a slot where other trends membrane domains containing z |
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0:02:10 | x three john g motifs can slide into each other in interact all so that |
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0:02:15 | are l two peptide or are l two region |
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0:02:19 | and then a mute of the l two that we created for the each team |
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0:02:23 | you |
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0:02:23 | the teaching and essentially just insert several very polar amino acids in to the middle |
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0:02:29 | of the trans membrane domain which otherwise is very non polar |
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0:02:34 | and what we found it is that the wild type l two trends member a |
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0:02:38 | domain doesn't fact function as at remembering domain but are each d you version does |
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0:02:43 | not so we now know that the |
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0:02:48 | defect in |
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0:02:49 | the h t mutant is that it can't escape the end result |
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0:02:54 | so |
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0:02:55 | we then we're and we wanted to |
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0:02:58 | study more closely those d x three d motifs and how they affect this process |
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0:03:02 | and so we created several alan insertion you switch unlike the h t new do |
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0:03:08 | not destroy to trans membrane functionalities of this region but what it will do is |
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0:03:13 | it'll throw out of key all those d x three g motifs |
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0:03:17 | and what we found with those means is that |
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0:03:20 | the one called eight fifty five was completely non infectious |
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0:03:25 | the a sixty was slightly infectious and the a sixty four was completely normal so |
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0:03:29 | this tells us the team or and terminology extra g might use are essential for |
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0:03:33 | infection the more see terminal ones or not |
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0:03:36 | we then went into a |
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0:03:39 | we then wanted to study |
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0:03:42 | what exactly those the extra g motifs are interacting with and interacting with themselves or |
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0:03:47 | their interacting with some unknown factor given the way h p v works most people |
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0:03:52 | clearly infection on their own |
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0:03:54 | and it doesn't become a problem joe years after the actual infection process is over |
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0:03:59 | it doesn't sir this peptide isn't really medically relevant |
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0:04:03 | two age pvi stopping h p v infection but we hope to take that same |
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0:04:08 | idea and applied to more medically relevant viruses that might use the same type of |
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0:04:14 | infection system |
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0:04:16 | and so |
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0:04:17 | in conclusion |
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0:04:19 | essentially what we found is that |
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0:04:21 | h p v gets a genome across that and is normal membrane by using a |
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0:04:26 | trend member a domain your the end terminus of l two which can change g |
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0:04:31 | x three g maltese along which the l two protein interacts with itself and perhaps |
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0:04:36 | other proteins form for that the genome can get through |
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